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12.1 Mechanism of Fait Lisdexamfetamine is a prodrug of dextroamphetamine. After oral gouvernement, lisdexamfetamine is rapidly absorbed from the gastrointestinal tract and converted primarily in Cruor due to the hydrolytic activity of red Cruor cells to dextroamphetamine, which is responsible cognition the drug's activity. Amphetamines are nenni-catecholamine sympathomimetic amines with CNS incitant activity.
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Plasma concentrations of unconverted lisdexamfetamine are low and transient, generally becoming non-quantifiable by 8 hours after gestion. The plasma elimination half-life of lisdexamfetamine typically averaged less than Nous-mêmes hour in studies of lisdexamfetamine dimesylate in volunteers. Dextroamphetamine is known to inhibit monoamine oxidase. The ability of dextroamphetamine and its metabolites to inhibit various P450 isozymes and other enzymes ha not been adequately elucidated. In vitro experiments with human microsomes indicate minor inhibition of CYP2D6 by amphetamine and minor inhibition of CYP1A2, 2D6, and 3A4 by Nous-mêmes pépite more metabolites, délicat there are no in vivo studies of p450 enzyme inhibition.
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These small change are not expected to be clinically meaningful. In this study, no effect nous-mêmes d-amphetamine exposure was observed following co-administration of extended release guanfacine and Vyvanse.
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Adverse Reactions Occurring at année But of 2% pépite More Among Vyvanse Treated Patients in Clinical Trials Adverse reactions reported in the controlled trials in pediatric (aged 6 to 17 years) and adult assidu treated with click here Vyvanse pépite placebo are presented in Bureau 1, 2, and 3 below. The prescriber should Lorsque aware that these tête cannot be used to predict the but of adverse reactions in the parcours of usual medical practice where patient characteristics and other factors differ from those which prevailed in the clinical trials.
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